A flower-extract study explicitly separated anticonvulsant observations from an unconfirmed mechanism.
Methanolic and acetone extracts protected mice in selected chemically induced seizure models but did not show the same effect against maximal electroshock. The authors proposed that flavonoids acting at the benzodiazepine site might contribute. They also stated that the study did not test that mechanism.
That qualification is central: an observed anticonvulsant effect is not direct evidence of receptor binding.
Different seizure models probe different processes. The lack of an electroshock effect limits a general antiepileptic claim, while results in other models remain preliminary. The extracts also contain multiple constituents.
For a faithful summary, distinguish the measured behavioral protection from the proposed molecular explanation. The paper does not establish clinical efficacy, a safe supplement-benzodiazepine combination, or a replacement for prescribed seizure treatment.
Original analysis of the supplied journal article, identified as PMC6470299.